Overview
- Editors:
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Luis Alfonso Rio
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Alain Puppo
- Presents recent advances in the field of reactive oxygen species (ROS) in plants highlighting new data and concepts
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Table of contents (14 chapters)
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- Izumi C. Mori, Yoshiyuki Murata, Misugi Uraji
Pages 25-42
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- Elizabeth Bell, Seiji Takeda, Liam Dolan
Pages 43-53
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- Jasmine Pham, Radhika Desikan
Pages 55-71
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- Alfonso Ros Barceló, V. Gómez Ros Laura
Pages 73-93
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- Luis A. del Río, Luisa M. Sandalio, Francisco J. Corpas, María C. Romero-Puertas, José M. Palma
Pages 95-111
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- G. Paul Bolwell, Arsalan Daudi
Pages 113-133
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- Karine Mandon, Nicolas Pauly, Alexandre Boscari, Renaud Brouquisse, Pierre Frendo, Bruce Demple et al.
Pages 135-147
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- Sandy Vanderauwera, Frank A. Hoeberichts, Frank Van Breusegem
Pages 149-164
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- Luisa M. Sandalio, María Rodríguez-Serrano, Luis A. del Río, María C. Romero-Puertas
Pages 175-189
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- Kirk Overmyer, Michael Wrzaczek, Jaakko Kangasjärvi
Pages 191-207
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- Françoise Simon-Plas, Sébastien Mongr
Pages 209-220
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Back Matter
Pages 241-245
About this book
Oxygen (O ) appeared in significant amounts in the Earth’s atmosphere over 2. 2 2 billion years ago, largely due to the evolution of photosynthesis by cyanobacteria (Halliwell 2006). The O molecule is a free radical, as it has two impaired electrons 2 that have the same spin quantum number. This spin restriction makes O prefer to 2 accept its electrons one at a time, leading to the generation of the so-called reactive oxygen species (ROS). The chemical nature of these species dictates that they can create damage in cells. This has contributed to the creation of the “oxidative stress” concept; in this view, ROS are unavoidable toxic products of O metabolism and 2 aerobic organisms have evolved antioxidant defences to protect against this tox- ity (Halliwell 1981; Fridovich 1998). Indeed, even in present-day plants, which are full of antioxidants, much of the protein synthetic activity of chloroplasts is used to replace oxidatively damaged D1 and other proteins (Halliwell 2006). Yet, the use of the “oxidative stress” term implies that ROS exert their effects through indiscriminate widespread inactivation of cellular functions. In this context, ROS must not be able to react with lipids, proteins or nucleic acids in order to avoid any damage to vital cellular components. However, genetic evidence has suggested that, in planta, purely physicoche- cal damage may be more limited than previously thought (Foyer and Noctor 2005).
