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Spinal Cord Injury

  • Book
  • © 1982

Overview

Editors:
  1. N. Eric Naftchi

    1. Institute of Rehabilitation Medicine, New York University Medical Center, USA

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Table of contents (20 chapters)

  1. Front Matter

    Pages i-xviii
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  2. Molecular Mechanisms of Acute Traumatic Spinal Cord Injury

    1. Front Matter

      Pages 1-1
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    2. Glial Fibrillary Acidic Protein (GFA) and Neuroglial Scarring, A Review

      • A. Bignami, D. Dahl
      Pages 3-14

    3. Positive and Negative Contrast Myelograph in Spinal Trauma

      • N. T. Pay, A. E. George, M. V. Benjamin, R. T. Bergeron, J. P. Lin, I. Kricheff
      Pages 15-28

    4. Effect of Pharmacological Agents on Normalization of Molecular and Histologic Dysfunction Following Traumatic Injury to the Spinal Cord

      • N. E. Naftchi, M. Demeny, H. Demopoulos, E. Flamm
      Pages 29-44

    5. Molecular Pathogenesis of Spinal Cord Degeneration After Traumatic Injury

      • H. B. Demopoulos, E. S. Flamm, M. L. Seligman, J. Ransohoff, N. E. Naftchi
      Pages 45-64

  3. Sequelae of Spinal Cord Injury: Possible Mechanisms of Spasticity and Pain Perception

    1. Front Matter

      Pages 65-65
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    2. Changes in the CNS Biogenic Amines and Tyrosine Hydroxylase Activity After Spinal Cord Transection in the Rat

      • N. E. Naftchi, A. K. Kirschner, M. Demeny, A. Viau
      Pages 67-80

    3. Brain Catecholamines, Serotonin, and Gamma-Aminobutyric Acid and Adrenal Catecholamines After High Spinal Cord Transection in the Rat

      • S. R. Snider
      Pages 81-84

    4. Substance P and Leucine-Enkephalin Changes in Spinal Cord of Paraplegic Rats and Cats

      • N. E. Naftchi, S. J. Abrahams, H. M. St. Paul, L. L. Vacca
      Pages 85-101

    5. Quarternary and Monoamine Imbalance After Spinal Transection (A Possible Mechanism of Spasticity)

      • V. Sahgal, V. Subarami, M. Rodriguez, C. Wurster
      Pages 103-114

    6. Alterations of Various Physiological and Biochemical Parameters During the Development of Spasticity in the Chronic Spinal Cat

      • W. Schlosser, S. Franco, G. Bautz, D. W. Horst, N. E. Naftchi
      Pages 115-131

    7. Clinical Trial of an Alpha Adrenergic Receptor Stimulating Drug (Clonidine) For Treatment of Spasticity in Spinal Cord Injured Patients

      • J. Tuckman, D. S. Chu, C. R. Petrillo, N. E. Naftchi
      Pages 133-137

  4. Hemodynamic Changes During Autonomic Dysreflexia

    1. Front Matter

      Pages 139-139
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    2. Relationship Between Serum Dopamine-β-Hydroxylase Activity, Catecholamine Metabolism, and Hemodynamic Changes During Paroxysmal Hypertension in Quadriplegia

      • N. E. Naftchi, G. F. Wooten, E. W. Lowman, J. Axelrod
      Pages 141-157

    3. Autonomic Hyperreflexia: Hemodynamics, Blood Volume, Serum Dopamine-β-Hydroxylase Activity, and Arterial Prostaglandin PGE2

      • N. E. Naftchi, M. Demeny, M. Berard, D. Manning, J. Tuckman
      Pages 159-176

  5. Bone Mineral and Matrix Changes Following Spinal Cord Injury: Possible Modes of Treatment

    1. Front Matter

      Pages 177-177
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    2. Disodium Etidronate in the Prevention of Postoperative Recurrence of Heterotopic Ossification in Spinal Cord Injured Patients

      • S. L. Stover, K. M. W. Niemann, J. M. Miller III
      Pages 179-192

    3. Reversal by Thyrocalcitonin of Depressed Calcium, Magnesium, and Phosphorus Balances in Paraplegic Rats

      • N. E. Naftchi, A. T. Viau, E. W. Lowman
      Pages 193-203

    4. Mineral Metabolism and the Effect of Thyrocalcitonin on Periarticular Bone in Spinal Cord Injured Man

      • N. E. Naftchi, A. T. Viau, G. H. Sell, E. W. Lowman
      Pages 205-217
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Keywords

About this book

We shall not, and those who come after us must not, accept the goals that were not reached yesterday as unsurmountable today or tomorrow. We will strive to render the world of the paralyzed-on-wheels but a transitory stop, and settle for nothing short of optimal recovery. N. Eric Naftchi In man, the process of "encephalization" culminates in almost complete control of the brain over the lower centers. Transection of the spinal cord severs the extensions of its nerve fiber tracts running to and from various brain centers. Although there is some confusion on the meaning of spinal shock, it is supposed to last from two to three weeks or longer in man, compared with less than a few minutes in the frog. This is a testimony to the complexity of the suprasegmental control in higher animals. Since the brain exerts its control over the internal environment through several monoamine, amino acids, and peptide neurotransmitters, it should not be surprising if the metabolism of these transmitters is found to be drastically altered along with other physical and metabolic dysfunctions which ensue following the spinal cord section. In spite of the major strides in rehabilitation of traumatic spinal cord injury, our knowledge of the etiology underlying the diverse neurophysiologic derangements remains limited. For instance, we are just becoming aware of some of the changes in the "milieu interieur.

Editors and Affiliations

  • Institute of Rehabilitation Medicine, New York University Medical Center, USA

    N. Eric Naftchi

Bibliographic Information

  • Book Title: Spinal Cord Injury

  • Editors: N. Eric Naftchi

  • DOI: https://doi.org/10.1007/978-94-011-6305-7

  • Publisher: Springer Dordrecht

  • eBook Packages: Springer Book Archive

  • Copyright Information: Spectrum Publications, Inc. 1982

  • Softcover ISBN: 978-94-011-6307-1Published: 13 February 2012

  • eBook ISBN: 978-94-011-6305-7Published: 06 December 2012

  • Edition Number: 1

  • Number of Pages: 269

  • Topics: Orthopedics

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