Skip to main content
SpringerLink
Log in

Cardiac Left Ventricular Hypertrophy

  • Book
  • © 1983

Overview

Editors:
  1. H. E. D. J. Keurs

    1. Department of Cardiology, Leiden University Hospital, Leiden, The Netherlands

    View editor publications

    You can also search for this editor in PubMed   Google Scholar

  2. J. J. Schipperheyn

    1. Department of Cardiology, Leiden University Hospital, Leiden, The Netherlands

    View editor publications

    You can also search for this editor in PubMed   Google Scholar

Part of the book series: Developments in Cardiovascular Medicine (DICM, volume 33)

This is a preview of subscription content, log in via an institution to check access.

Access this book

Log in via an institution
eBook USD 39.99
Price excludes VAT (USA)
  • Available as EPUB and PDF
  • Read on any device
  • Instant download
  • Own it forever
Softcover Book USD 54.99
Price excludes VAT (USA)
  • Compact, lightweight edition
  • Dispatched in 3 to 5 business days
  • Free shipping worldwide - see info

Tax calculation will be finalised at checkout

Other ways to access

Licence this eBook for your library

Institutional subscriptions

Table of contents (17 chapters)

  1. Front Matter

    Pages I-XIV
    Download chapter PDF

  2. Prevalence and Risks of Left Ventricular Hypertrophy

    1. Front Matter

      Pages 1-1
      Download chapter PDF

    2. Epidemiologic Features of Left Ventricular Hypertrophy in Normotensive and Hypertensive Subjects

      • D. D. Savage, R. D. Abbott, S. Padgett, S. J. Anderson, R. J. Garrison
      Pages 2-15

    3. Sensitivity of Echocardiography for Detection of Left Ventricular Hypertrophy

      • R. B. Devereux, D. R. Alonso, E. M. Lutas, T. G. Pickering, G. A. Harshfield, J. H. Laragh
      Pages 16-37

    4. Determinants and Modulators of Left Ventricular Structure

      • F. H. Messerli
      Pages 38-50

  3. Etiology and Functional Aspects at the Cellular Level

    1. Front Matter

      Pages 51-51
      Download chapter PDF

    2. Relationship Between Cardiac Work and Cardiac Growth: Some General Thoughts on Cardiac Hypertrophy

      • W. F. H. M. Mommaerts
      Pages 52-64

    3. Myocardial cell Properties and Hypertrophy

      • H. E. D. J. Keurs, B. J. M. Ter Mulder, V. J. A. Schouten
      Pages 66-88

  4. Etiology and functional aspects of the hypertrophied heart

    1. Front Matter

      Pages 89-89
      Download chapter PDF

    2. Some Functional Consequences of Left Ventricular Hypertrophy in Hypertension

      • R. C. Tarazi
      Pages 90-101

    3. Left Ventricular Hypertrophy, Matching Between Heart and Arterial System

      • G. Elzinga
      Pages 102-113

    4. Assessment of the Function of the Hypertrophied Heart

      • F. M. Fouad
      Pages 114-128

  5. The Coronary Circulation in the Hypertrophied Heart

    1. Front Matter

      Pages 129-129
      Download chapter PDF

    2. Total and Transmural Perfusion of the Hypertrophied Heart

      • J. I. E. Hoffman, M. T. Grattan, F. L. Hanley, L. M. Messina
      Pages 130-151

    3. Left Ventricular Hypertrophy and Myocardial Ischemia

      • J. J. Schipperheyn, J. Jonas
      Pages 152-169

    4. Coronary Flow Mechanics of the Hypertrophied Heart

      • J. A. E. Spaan, P. Bruinsma, J. D. Laird
      Pages 170-193

  6. Diagnostic Development in Detection of Left Ventricular Hypertrophy

    1. Front Matter

      Pages 195-195
      Download chapter PDF

    2. Electrocardiographic Aspects of Left Ventricular Hypertrophy

      • H. E. Kulbertus
      Pages 196-207

    3. Radioisotope Studies in Patients With Left Ventricular Hypertrophy

      • G. A. Beller
      Pages 208-222

  7. Therapeutic Aspects of Left Ventricular Hypertrophy

    1. Front Matter

      Pages 223-223
      Download chapter PDF
Back to top

Keywords

About this book

Left ventricular hypertrophy (LVH) is usually considered to be a compen­ satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria.

Editors and Affiliations

  • Department of Cardiology, Leiden University Hospital, Leiden, The Netherlands

    H. E. D. J. Keurs, J. J. Schipperheyn

Bibliographic Information

  • Book Title: Cardiac Left Ventricular Hypertrophy

  • Editors: H. E. D. J. Keurs, J. J. Schipperheyn

  • Series Title: Developments in Cardiovascular Medicine

  • DOI: https://doi.org/10.1007/978-94-009-6759-5

  • Publisher: Springer Dordrecht

  • eBook Packages: Springer Book Archive

  • Copyright Information: Martinus Nijhoff Publishers, Boston 1983

  • Softcover ISBN: 978-94-009-6761-8Published: 11 November 2011

  • eBook ISBN: 978-94-009-6759-5Published: 06 December 2012

  • Series ISSN: 0166-9842

  • Edition Number: 1

  • Number of Pages: 300

  • Topics: Cardiology

Publish with us

Policies and ethics

Back to top

Search

Navigation