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Hypertrophic Cardiomyopathy

The Therapeutic Role of Calcium Antagonists

  • Conference proceedings
  • © 1982

Overview

Editors:
  1. M. Kaltenbach

    1. Zentrum der Inneren Medizin, Abteilung für Kardiologie, Klinikum der Johann-Wolfgang-von-Goethe-Universität, Frankfurt/Main 70, Germany

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  2. S. E. Epstein

    1. Cardiology Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, USA

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Table of contents (34 papers)

  1. Front Matter

    Pages I-XIV
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  2. Clinical and Anatomical Characterization of Hypertrophic Cardiomyopathy

    1. Front Matter

      Pages 1-1
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    2. Synopsis

      • J. F. Goodwin
      Pages 3-4

    3. Hypertrophic Cardiomyopathy: An Overview

      • Stephen E. Epstein, Barry J. Maron
      Pages 5-17

    4. Echocardiographic Identification of Patterns of Left Ventricular Hypertrophy in Hypertrophic Cardiomyopathy

      • Barry J. Maron, John S. Gottdiener, Stephen E. Epstein
      Pages 18-37

    5. Distribution and Significance of Cardiac Muscle Cell Disorganization in the Left Ventricle of Patients with Hypertrophic Cardiomyopathy: Evidence of a Diffuse Cardiomyopathic Process

      • Barry J. Maron, William C. Roberts
      Pages 38-57

    6. Left Ventricular Biopsy in Hypertrophic Cardiomyopathy: Light and Electron Microscopic Evaluations

      • B. Kunkel, M. Schneider, R. Hopf, G. Kober, K. Hübner, M. Kaltenbach
      Pages 58-69

  3. Cardiomyopathy in Animals and Therapeutic Interventions

    1. Front Matter

      Pages 71-71
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    2. Synopsis

      • Barry J. Maron
      Pages 72-72

    3. Spontaneously Occurring Hypertrophic Cardiomyopathy in Dogs and Cats: A Potential Animal Model of a Human Disease

      • Barry J. Maron, Si-Kwang Liu, Lawrence P. Tilley
      Pages 73-87

    4. Cardiac Effects of Nerve Growth Factor in Dogs

      • Michael P. Kaye, David J. Witzke, David J. Wells, Valentin Fuster
      Pages 88-98

    5. Prevention of Myocardial Cell Necrosis in the Syrian Hamster — Results of Long-Term Treatment

      • K. Lossnitzer, A. Konrad, D. Zeyer, W. Mohr
      Pages 99-114

    6. Prevention by Verapamil of Isoproterenol-Induced Hypertrophic Cardiomyopathy in Rats

      • A. Fleckenstein, M. Frey, J. Keidel
      Pages 115-120

  4. Effects of Acute Administration of Verapamil in Patients with Hypertrophic Cardiomyopathy

    1. Front Matter

      Pages 121-121
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    2. Synopsis

      • S. E. Epstein
      Pages 122-123

    3. Acute Hemodynamic Effects of Verapamil in Hypertrophic Cardiomyopathy

      • Douglas R. Rosing, Kenneth M. Kent, Robert O. Bonow, Stephen E. Epstein
      Pages 124-137

    4. Hemodynamics and Contractility After Oral, Intravenous, and Intracoronary Application of Calcium Antagonists

      • Wulf-Dirk Bussmann, Rüdiger Hopf, Alexander Trompler, Martin Kaltenbach
      Pages 138-147

    5. Effect of Verapamil on Left Ventricular Isovolumic Relaxation Time and Regional Left Ventricular Filling in Hypertrophic Cardiomyopathy

      • Peter Hanrath, Detlef G. Mathey, Peter Kremer, Frank Sonntag, Walter Bleifeld
      Pages 148-157

  5. Treatment of Hypertrophic Cardiomyopathy with Verapamil

    1. Front Matter

      Pages 159-159
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    2. Synopsis

      • H. Kuhn
      Pages 160-162
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Keywords

About this book

First described in 1907 by Schicke but recognized as a clinical entity only as recently as 1958, when Teare published the pathologic findings in patients with hypertrophic cardiomyopathy (HeM), an explosion of knowledge about this fascinating disease has occurred, which has caused a profound evolution of our understanding of its broad pathophysiologic and clinical spectrum. Progress has been particularly rapid in the past few years when M-mode echocardiography, and more recently 2-dimen­ sional echocardiography have been applied to the study ofHCM. In addition to new insights as to what the disease is, there has been enormous progress concerning its treatment, with the application of beta-adrenergic blocking agents and surgical relief of left ventricular outflow tract obstruction. Although these approaches have led to great strides in the symptomatic control of the disease, many patients' symptoms have remained refactory to medical and surgical therapy. Most discouragingly, sudden death still occurs, even in patients on large doses of beta-blocking agents and in patients who have had surgical relief of left ventricular outflow tract obstruction.

Editors and Affiliations

  • Zentrum der Inneren Medizin, Abteilung für Kardiologie, Klinikum der Johann-Wolfgang-von-Goethe-Universität, Frankfurt/Main 70, Germany

    M. Kaltenbach

  • Cardiology Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, USA

    S. E. Epstein

Bibliographic Information

  • Book Title: Hypertrophic Cardiomyopathy

  • Book Subtitle: The Therapeutic Role of Calcium Antagonists

  • Editors: M. Kaltenbach, S. E. Epstein

  • DOI: https://doi.org/10.1007/978-3-642-68282-7

  • Publisher: Springer Berlin, Heidelberg

  • eBook Packages: Springer Book Archive

  • Copyright Information: Springer-Verlag Berlin Heidelberg 1982

  • Softcover ISBN: 978-3-642-68284-1Published: 07 December 2011

  • eBook ISBN: 978-3-642-68282-7Published: 06 December 2012

  • Edition Number: 1

  • Number of Pages: XIV, 336

  • Topics: Cardiology, Pharmacology/Toxicology

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