after heated and often bitter debates, SIEBENMANN'S opinion finally prevailed, i. e. , a contribution to cochlear lesions due to vibrations of the floor transmitted via bone conduction could not be demonstrated. For one thing, it was hard to see how appreciable amounts of energy could reach the ears in this manner, considering the attenuation that is bound to occur across each of the many joints along the pathway involved. In some older audiological surveys conducted in industry (e. g. , TEMKIN, 1933), groups of workmen were found who displayed signs of apical-turn lesions, i. e. , low-tone hearing losses for air and for bone. Such lesions could not be expected to results from exposure to air-borne sounds because of the low-frequency attenu ation of the middle ear. Although WITTMAACK'S explanation, which was frequently invoked in such reports, does no longer appear tenable, such apical-turn lesions could conceivably be caused by bone conduction components of high-intensity noise in the sense of BEKESY (1948). - As far as I am aware of, no newer studies have been conducted in this problem area, and the older experiments and/or surveys were done at times before signal parameters could be precisely controlled or measured. A detailed, critical review of the older studies on the potential contribution of bone-conducted energy to industrial hearing loss and its underlying pathology may be found in Werner (1940) who, incidently, favored SIEBENMANN'S point of VIew.
Authors, Editors and Affiliations
Institut für Physiologie und Biokybernetik der Universität 8520 Erlangen, Germany
Wolf D. Keidel
I. Physiologisches Institut der Universität, Erlangen, Germany
Wolf D. Keidel
Center for Neural Sciences and Department of Psychology, Indiana University, Bloomington, USA
William D. Neff
Physical Laboratory, ENT Department, Wilhelmina Hospital, University of Amsterdam, Amsterdam - Oud Wes, The Netherlands
E. Boer
Tracor Inc., Austin, USA
W. K. Connor,
W. Rudmose
Central Institute for the Deaf, St. Louis, USA
H. Davis
E.N.T.-Department, Academisch Ziekenhuis, Leiden, The Netherlands
J. J. Eggermont
Department of Neurosciences, University of California at San Diego, La Jolla, USA
R. Galambos,
E. Hawkins
Department of Neurophysiology, University of Wisconsin, Medical School, Madison, USA
C. D. Geisler
The Callier Center for Communication Disorders, The University of Texas at Dallas, Dallas, USA
G. M. Gerken
Yellow Springs, USA
H. E. Gierke
Department of the Air Force, 6570th Aerospace Medical Research Laboratory (AFSC), Wright-Patterson Air Force Base, USA
H. E. Gierke
West Moors, Dorset, Ferndown 87, Great Britain
C. S. Hallpike
Kresge Hearing Research Institute, Medical School, The University of Michigan, Ann Arbor, USA
S. A. Hillyard
Institut für Physiologie und Biokybernetik, Universität Erlangen, Erlangen, Germany
W. D. Keidel
Department of Psychology, Miami University, Oxford, USA
D. E. Parker
Hospital General D’Ottawa, University of Ottawa, Ottawa, Canada
T. W. Picton
Division of Otolaryngology, Stanford University, Palo Alto, USA
F. B. Simmons
Hals-Nasen-Ohrenklinik, Universität Freiburg, 78 Freiburg, Germany
G. Stange
Department of Aeronautics and Astronautics, Stanford University, Stanford, USA
C. R. Steele
College of Physicians and Surgeons, Columbia University, New York, USA
J. Tonndorf
Hals-Nasen-Ohrenklinik, Universität Erlangen, Erlangen, Germany
M. E. Wigand
Hals-Nasen-Ohrenklinik, Universität Freiburg, Freiburg, Germany
F. Zöllner
Bibliographic Information
Book Title: Auditory System
Book Subtitle: Clinical and Special Topics
Authors: E. Boer, W. K. Connor, H. Davis, J. J. Eggermont, R. Galambos, C. D. Geisler, G. M. Gerken, H. E. Gierke, C. S. Hallpike, E. Hawkins, S. A. Hillyard, W. D. Keidel, D. E. Parker, T. W. Picton, W. Rudmose, F. B. Simmons, G. Stange, C. R. Steele, J. Tonndorf, M. E. Wigand, … F. Zöllner
