Editors:
- The unique feature of this book is its comparative approach to different genetic model systems
- Full text available online for subscribers of the series
Part of the book series: Topics in Current Genetics (TCG, volume 3)
Buy it now
Buying options
Tax calculation will be finalised at checkout
Other ways to access
This is a preview of subscription content, log in via an institution to check for access.
Table of contents (10 chapters)
-
Front Matter
-
Back Matter
About this book
Aging is the progressive decline in biological functions over time. This decline targets macromolecules, cells, tissues and, as a consequence, whole organisms. Despite considerable progress in the development of testable hypothesis concerning aging in an evolutionary context, a unifying theory of the molecular/physiological mechanistic causes of aging has not been reached. In fact, is it not clear to what extent aging is a programmed or stochastic process.
This book takes the reader from unicellular bacterial deterioration via senescence in fungi and worms to aging in rodents and humans, allowing a comparative view on similarities and differences in different genetic model systems. The different model systems are scrutinized in the light of contemporary aging hypothesis, such as the free radical and genomic instability theories.
Editors and Affiliations
-
Dept. Cell & Molecular Biology, Göteborg University, Göteborg, Sweden
Thomas Nyström
-
und Biotechnologie Botanisches Institut, Univ. Frankfurt Molekulare Entwicklungsbiol., Frankfurt, Germany
Heinz D. Osiewacz
Bibliographic Information
Book Title: Model Systems in Aging
Editors: Thomas Nyström, Heinz D. Osiewacz
Series Title: Topics in Current Genetics
DOI: https://doi.org/10.1007/978-3-540-37005-5
Publisher: Springer Berlin, Heidelberg
-
eBook Packages: Springer Book Archive
Copyright Information: Springer-Verlag Berlin Heidelberg 2004
Hardcover ISBN: 978-3-540-02490-3Published: 22 September 2003
Softcover ISBN: 978-3-642-05698-7Published: 22 October 2010
eBook ISBN: 978-3-540-37005-5Published: 24 March 2011
Series ISSN: 1610-2096
Series E-ISSN: 1610-6970
Edition Number: 1
Number of Pages: XV, 301
Topics: Cell Biology, Biomedicine general, Developmental Biology