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Oxidative Stress and Neuroprotection

  • Book
  • © 2006

Overview

Editors:
  1. H. Parvez

    1. Institute Alfred Fessard de Neurosciences, CNRS, Gif Yvette, France

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  2. P. Riederer

    1. Klinik und Poliklinik für Psychiatrie und Psychotherapie, Würzburg, Germany

    View editor publications

    You can also search for this editor in PubMed   Google Scholar

Part of the book series: Journal of Neural Transmission. Supplementa (NEURALTRANS, volume 71)

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Table of contents (26 chapters)

  1. Front Matter

    Pages I-XIV
    Download chapter PDF

  2. Levodopa in the treatment of Parkinson’s disease

    • S. Fahn
    Pages 1-15

  3. Changing dopamine agonist treatment in Parkinson’s disease: experiences with switching to pramipexole

    • H. Reichmann, P. Odin, H. M. Brecht, J. Köster, P. H. Kraus
    Pages 17-25

  4. The DONPAD-study — Treatment of dementia in patients with Parkinson’s disease with donepezil

    • T. Muüller, J. Welnic, G. Fuchs, H. Baas, G. Ebersbach, H. Reichmann
    Pages 27-30

  5. PD-related psychosis: pathophysiology with therapeutical strategies

    • E. Ch. Wolters
    Pages 31-37

  6. Antioxidant capacity in postmortem brain tissues of Parkinson’s and Alzheimer’s diseases

    • E. Sofic, A. Sapcanin, I. Tahirovic, I. Gavrankapetanovic, K. Jellinger, G. P. Reynolds et al.
    Pages 39-43

  7. Apoptosis inhibition in T cells triggers the expression of proinflammatory cytokines — implications for the CNS

    • C. Scheller, P. Riederer, M. Gerlach, E. Koutsilieri
    Pages 45-51

  8. Molecular mechanism of the relation of monoamine oxidase B and its inhibitors to Parkinson’s disease: possible implications of glial cells

    • T. Nagatsu, M. Sawada
    Pages 53-65

  9. Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling leading to cell death or neuroprotection

    • M. Naoi, W. Maruyama, Y. Akao, H. Yi, Y. Yamaoka
    Pages 67-77

  10. The relationship of early studies of monoamine oxidase to present concepts

    • I. J. Kopin
    Pages 79-86

  11. Isatin, an endogenous MAO inhibitor, and a rat model of Parkinson’s disease induced by the Japanese encephalitis virus

    • M. Minami, N. Hamaue, M. Hirafuji, H. Saito, T. Hiroshige, A. Ogata et al.
    Pages 87-95

  12. Isatin interaction with glyceraldehyde-3-phosphate dehydrogenase, a putative target of neuroprotective drugs: partial agonism with deprenyl

    • A. Medvedev, O. Buneeva, O. Gnedenko, V. Fedchenko, M. Medvedeva, Y. Ivanov et al.
    Pages 97-103

  13. Inhibition of amine oxidases by the histamine-1 receptor antagonist hydroxyzine

    • J. O’Sullivan, M. I. O’Sullivan, K. F. Tipton, G. Davey
    Pages 105-112

  14. Neuroprotection for Parkinson’s disease

    • P. A. LeWitt
    Pages 113-122

  15. Marker for a preclinical diagnosis of Parkinson’s disease as a basis for neuroprotection

    • Daniela Berg
    Pages 123-132

  16. Assessing neuroprotection in Parkinson’s disease: from the animal models to molecular neuroimaging in vivo

    • R. Ceravolo, P. Sgadò, D. Frosini, G. U. Corsini
    Pages 133-141

  17. Deprenyl: from chemical synthesis to neuroprotection

    • K. Magyar, M. Pálfi, V. Jenei, É. Szökő
    Pages 143-156

  18. The use of rasagiline in Parkinson’s disease

    • A. H. V. Schapira
    Pages 157-161

  19. Novel neuroprotective neurotrophic NAP analogs targeting metal toxicity and oxidative stress: potential candidates for the control of neurodegenerative diseases

    • H. Zheng, D. Blat, M. Fridkin
    Pages 163-172

  20. Acute and chronic effects of developmental iron deficiency on mRNA expression patterns in the brain

    • S. L. Clardy, X. Wang, W. Zhao, W. Liu, G. A. Chase, J. L. Beard et al.
    Pages 173-196
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Keywords

About this book

This book deals with basic and clinical aspects of monoamine oxidase (MAO) subtypes A and B highlighting its importance in neurological and psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson’s disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induced oxidative stress in various experimental studies and clinical conditions. Based on these findings, new compounds have been developed which not only block MAO, but are in addition, either inhibitors of acetylcholine esterase or have iron chelating properties. The aspect of "preclinical" and "clinical" neuro protection as well as MAO neuroprotection are additional topics covered in this book. MAO, iron and neuroprotection are seen in the framework of general anti Parkinson’s therapy with chapters on levodopa, dopaminergic receptor agonists and clinical issues.

Editors and Affiliations

  • Institute Alfred Fessard de Neurosciences, CNRS, Gif Yvette, France

    H. Parvez

  • Klinik und Poliklinik für Psychiatrie und Psychotherapie, Würzburg, Germany

    P. Riederer

Bibliographic Information

  • Book Title: Oxidative Stress and Neuroprotection

  • Editors: H. Parvez, P. Riederer

  • Series Title: Journal of Neural Transmission. Supplementa

  • DOI: https://doi.org/10.1007/978-3-211-33328-0

  • Publisher: Springer Vienna

  • eBook Packages: Medicine, Medicine (R0)

  • Copyright Information: Springer-Verlag Vienna 2006

  • Hardcover ISBN: 978-3-211-33327-3Published: 15 September 2006

  • Softcover ISBN: 978-3-7091-1736-1Published: 28 November 2014

  • eBook ISBN: 978-3-211-33328-0Published: 26 December 2006

  • Series ISSN: 0303-6995

  • Edition Number: 1

  • Number of Pages: XIV, 264

  • Additional Information: Special edition of "Journal of Neural Transmission", Suppl. 71

  • Topics: Neurology

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