Cellular Interactions in Cardiac Pathophysiology

1. Front Matter

   Pages i-1

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2. Extracellular matrix and cardiocyte interaction

   1. Front Matter

      Pages 3-3

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   2. Role of early reperfusion in the induction of adhesion molecules and cytokines in previously ischemic myocardium

      • Gilbert L. Kukielka, Keith A. Youker, Lloyd H. Michael, Ajith G. Kumar, Christie M. Ballantyne, C. Wayne Smith et al.

      Pages 5-12

   3. Importance of monocytes/macrophages and fibroblasts for healing of micronecroses in porcine myocardium

      • Dorothée Weihrauch, Margarete Arras, René Zimmermann, Jutta Schaper

      Pages 13-19

   4. Cell-cell and cell-matrix adhesion molecules in human heart and lung transplants

      • Gustav Steinhoff, Axel Haverich

      Pages 21-27

   5. Progression of heart failure: A role for interstitial fibrosis

      • Hani N. Sabbah, Victor G. Sharov, Michael Lesch, Sidney Goldstein

      Pages 29-34

   6. Chronic hibernating myocardium: Interstitial changes

      • Jannie Ausma, Jack Cleutjens, Fred Thoné, Willem Flameng, Frans Ramaekers, Marcel Borgers

      Pages 35-42

   7. Differences between atrial and ventricular protein profiling in children with congenital heart disease

      • Václav Pelouch, Marie Milerová, Bohuslav Ošťádal, Bohumil Hučín, Milan Šamánek

      Pages 43-49

   8. Ventricular remodeling: insights from pharmacologic interventions with angiotensin-converting enzyme inhibitors

      • Sidney Goldstein, Victor G. Sharov, Jane M. Cook, Hani N. Sabbah

      Pages 51-55

   9. Factors involved in capillary growth in the heart

      • Olga Hudlická, Margaret D. Brown, Helene Walter†, Jacqueline B. Weiss, Anita Bate

      Pages 57-68

   10. Biomechanical signals in the coronary artery triggering the metabolic processes during cardiac overload

       • Mária Gerová, Olga Pecháňová, Venceslav Stoev, Margita Kittová, Iveta Bernátová, Milan Juráni et al.

       Pages 69-73

3. Myocytic adaptation and myocardial injury

   1. Front Matter

      Pages 75-75

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   2. Oxidative stress and heart failure

      • Neelam Singh, Arvinder K. Dhalla, Charita Seneviratne, Pawan K. Singal

      Pages 77-81

   3. Structural and biochemical remodelling in catecholamine-induced cardiomyopathy: comparative and ontogenetic aspects

      • Bohuslav Ošťádal, Václav Pelouch, Ivana Ošťádalová, Olga Nováková

      Pages 83-88

   4. Angiotensin converting enzyme inhibitors, left ventricular hypertrophy and fibrosis

      • Wolfgang Linz, Gabriele Wiemer, Jutta Schaper, René Zimmermann, Kazushige Nagasawa, Peter Gohlke et al.

      Pages 89-97

   5. Influence of global ischemia on the sarcolemmal ATPases in the rat heart

      • Norbert Vrbjar, Andrej Džurba, Attila Ziegelhöffer

      Pages 99-103

   6. Response of the rat heart to catecholamines and thyroid hormones

      • Heinz-Gerd Zimmer, Michael Irlbeck, Claudia Kolbeck-Rühmkorff

      Pages 105-114

   7. On the mechanism and possible therapeutic application of delayed adaptation of the heart to stress situations

      • Ernst-Georg Krause, László Szekeres

      Pages 115-122

   8. Ischaemic preconditioning in the rat heart: The role of G-proteins and adrenergic stimulation

      • Tanya Ravingerová, Nigel J. Pyne, James R. Parratt

      Pages 123-128

   9. Adaptation of the heart to ischemia by preconditioning: Effects on energy equilibrium properties of sarcolemmal ATPases and release of cardioprotective proteins

      • Attila Ziegelhöffer, Norbert Vrbjar, Ján Styk, Albert Breier, Andrej Džurba, Tatjana Ravingerová

      Pages 129-137

Despite the considerable success in treating diseases of the heart and blood vessels, they still remain the major cause of mortality throughout the world. One of the reasons underlying this problem is our lack of understanding of the molecular and cellular aspects of the processes involved.
These problems are fully discussed in Cellular Interactions in Cardiac Pathophysiology, which draws together 25 contributions from leading investigators from all parts of the world. The contributions are grouped under three headings: Extracellular matrix and cardiocyte interaction; Myocytic adaptations and myocardial injury; and Signal transduction.

• ATP
• arrhythmia
• artery
• congenital heart disease
• cytokine
• enzyme
• enzymes
• heart
• heart disease
• heart failure
• pharmacology
• physiology
• protein
• proteins
• receptor

• Institute for Heart Research, Slovak Academy of Sciences, Bratislava, Slovak Republic

  Ján Slezák, Attila Ziegelhöffer

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