Cell Volume and Signaling

1. Front Matter

   Pages i-xxiii

   PDF

2. Transport in avian red cells

   1. The Balancing Act of the Naked Cell

      • John S. Willis

      Pages 1-9

3. Electroneutral Cotransporters

   1. Twenty-Five Years of K-Cl Cotransport: From Stimulation by a Thiol Reaction to Cloning of the Full-Length KCCs

      • Peter K. Lauf, Norma C. Adragna

      Pages 11-28

   2. Molecular Physiology of Mammalian K+-CL− Cotransporters

      • Adriana Mercado, Gerardo Gamba, David B. Mount

      Pages 29-41

   3. STE20 Kinases and Cation-Chloride Cotransporters

      • Eric Delpire, Kerstin Piechotta

      Pages 43-53

   4. Molecular Physiology of the Renal Na+-Cl− and Na+-K+-2Cl− Cotransporters

      • Gerardo Gamba, Norma A. Bobadilla

      Pages 55-65

   5. The Role of the Blood-Brain Barrier Na-K-2Cl Cotransporter in Stroke

      • Martha E. O’Donnell, Tina I. Lam, Lien Tran, Steven E. Anderson

      Pages 67-75

   6. Regulation of Na-K-2Cl Cotransport in Red Cells

      • Peter W Flatman

      Pages 77-88

   7. A Novel NHE1 from Red Blood Cells of the Winter Flounder

      • Stine Falsig Pedersen

      Pages 89-98

4. Ion Channels and Exchangers

   1. Probing of the ICln Channel Pore by Cysteine Mutagenesis and Cadmium-Block

      • M. Jakab, M. L. Garavaglia, J. Fürst, S. Rodighiero, F. Guizzardi, G. Meyer et al.

      Pages 99-108

   2. Volume-Dependent and -Independent Activated Anion Conductances and Their Interaction in the Renal Inner Medullary Collecting Duct (IMCD)

      • Stefan H. Boese, Mike A. Gray, Nick L. Simmons

      Pages 109-118

   3. Secretory Control of Basolateral Membrane Potassium and Chloride Channels in Colonic Crypt Cells

      • Dan R. Halm

      Pages 119-129

   4. Effects of Ammonium on Ion Channels and Transporters in Colonic Secretory Cells

      • Roger T. Worrell, Jeffrey B. Matthews

      Pages 131-139

   5. The Volume-Activated Chloride Current Depends on Phospholipase C Activation and Intracellular Calcium Mobilization

      • D. Varela, F. Simon, A. Riveros, F. Jørgensen, A. Stutzin

      Pages 142-145

   6. Nitric Oxide (NO) Modulation of Cl-Dependent Transporters in the Kidney

      • Pablo A. Ortiz, Jeffrey L. Garvin

      Pages 147-156

   7. Volume Activated Anion Channel and Astrocytic Cellular Edema in Traumatic Brain Injury and Stroke

      • Harold K. Kimelberg

      Pages 157-167

5. Cell Volume, Cytoskeleton and Apoptosis

   1. Effectors and Signaling Events Activated by Cell Shrinkage in Ehrlich Ascites Tumor Cells

      • Else K. Hoffmann, Stine Falsig Pedersen

      Pages 169-178

   2. Water Movement During Apoptosis

      • Elizabeth Jablonski, Ashley Webb, Francis M. Hughes Jr.

      Pages 179-188

   3. Apoptosis and Cell Volume Regulation

      • Gerd Heimlich, Carl D. Bortner, John A. Cidlowski

      Pages 189-203

   4. Anion Channel Involved in Induction of Apoptosis and Necrosis

      • Yasunobu Okada, Emi Maeno, Shin-ichiro Mori

      Pages 205-209

In front of you is the finished product of your work, the text of your contributions to the 2003 Dayton International Symposium on Cell Volume and Signal Transduction. As we all recall, this symposium brought together the Doyens of Cellular and Molecular Physiology as well as aspiring young investigators and students in this field. It became a memorable event in an illustrious series of International Symposia on Cell Volume and Signaling. This series, started by Professors Vladimir Strbák, Florian Lang and Monte Greer in Smolenice, Slovakia in 1997 and continued by Professors Rolf Kinne, Florian Lang and Frank Wehner in Berlin in 2000, is projected for 2005 in Copenhagen to be hosted by our colleague, Professor Else Hoffmann and her team. We dearly miss Monte Greer to whom this symposium was dedicated and addressed so eloquently by Vladimir Strbák in his Dedication to Monte. Monte and I became friends in Smolenice and had begun to discuss the 2003 meeting only a few days before his tragic accident in 2002. There are others who were not with us, and we missed them, too. We would not have been able to succeed in this event without the unflagging support of our higher administration at Wright State University, the NIDDKD of the National Institute of Health, and the Fuji Medical System (see Acknowledgments).

• Activation
• blood cell
• cells
• erythrocyte
• genes
• pathophysiology
• physiology
• red blood cells
• regulation
• signal transduction
• skeletal muscle

• Wright State University, Dayton

  Peter K. Lauf, Norma C. Adragna

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