Innate Immune Response and Inflammasome Activation During SARS-CoV-2 Infection
Authors (first, second and last of 6)
Inflammation presents the latest international advances in experimental and clinical research on the physiology, biochemistry, cell biology, and pharmacology of inflammation. Published items include full-length scientific reports, short definitive articles, and papers from meetings and symposia proceedings. The journal's coverage extends to acute and chronic inflammation; mediators of inflammation; mechanisms of tissue injury and cytotoxicity; pharmacology of inflammation; and clinical studies of inflammation and its modification.
Details Concerning the Submission and Publication Procedures:
- No Page Charges
- No Fees for Online Color Images
- Optional Color Images in Print – EUR 950/USD 1150. VAT or local taxes will be added where applicable.
- Optional Open Access Publication Fee (APC) - EUR 2990/USD 3760/GBP 2490. VAT or local taxes will be added where applicable.
- Reports on the latest international advances in understanding and treating inflammation
- Covers acute and chronic inflammation, pharmacology, cytotoxicology and among other topics
- Offers papers, short articles, proceedings and more
- 100% of authors who answered a survey reported that they would definitely publish or probably publish in the journal again
Journal information
- Editor-in-Chief
-
- Massimo Gadina
- Publishing model
- Hybrid (Transformative Journal). How to publish with us, including Open Access
Journal metrics
- 4.657 (2021)
- Impact factor
- 4.361 (2021)
- Five year impact factor
- 7 days
- Submission to first decision (Median)
- 368,197 (2021)
- Downloads
Latest issue
Latest articles
-
-
NLRP3 Inflammasome in Atherosclerosis: Putting Out the Fire of Inflammation
Authors (first, second and last of 6)
-
Convergence of Fructose-Induced NLRP3 Activation with Oxidative Stress and ER Stress Leading to Hepatic Steatosis
Authors (first, second and last of 8)
-
Pectolinarigenin Suppresses LPS-Induced Inflammatory Response in Macrophages and Attenuates DSS-Induced Colitis by Modulating the NF-κB/Nrf2 Signaling Pathway
Authors (first, second and last of 5)
-
PCSK9 Promotes Endothelial Dysfunction During Sepsis Via the TLR4/MyD88/NF-κB and NLRP3 Pathways
Authors (first, second and last of 6)
About this journal
- Electronic ISSN
- 1573-2576
- Print ISSN
- 0360-3997
- Abstracted and indexed in
-
- ANVUR
- BFI List
- BIOSIS
- Baidu
- Biological Abstracts
- CAB Abstracts
- CLOCKSS
- CNKI
- CNPIEC
- Chemical Abstracts Service (CAS)
- Current Contents/ Life Sciences
- Dimensions
- EBSCO Academic Search
- EBSCO Biomedical Reference Collection
- EBSCO Discovery Service
- EBSCO STM Source
- EMBASE
- EMBiology
- Google Scholar
- IFIS Publishing
- Japanese Science and Technology Agency (JST)
- Journal Citation Reports/Science Edition
- Medline
- Meta
- Naver
- Norwegian Register for Scientific Journals and Series
- OCLC WorldCat Discovery Service
- Pathway Studio
- Portico
- ProQuest-ExLibris Primo
- ProQuest-ExLibris Summon
- Reaxys
- SCImago
- SCOPUS
- Science Citation Index
- Science Citation Index Expanded (SCIE)
- TD Net Discovery Service
- UGC-CARE List (India)
- Wanfang
- Copyright information
-
© Springer Science+Business Media, LLC, part of Springer Nature
