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T Cells in Arthritis

  • Book
  • © 1998

Overview

Editors:
  1. Pierre Miossec

    1. Departments of Immunology and Rheumatology Hôpital Edouard Herriot, University Claude Bernard, Lyon Cedex 03, France

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  2. Gary S. Firestein

    1. Division of Rheumatology, UCSD School of Medicine, San Diego, USA

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  3. Wim B. Berg

    1. Department of Rheumatology, University Hospital Nijmegen, Nijmegen, The Netherlands

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Part of the book series: Progress in Inflammation Research (PIR)

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Table of contents (11 chapters)

  1. Front Matter

    Pages i-xii
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  2. T cells as secondary players in rheumatoid arthritis

    • Katherine H. Y. Nguyen, Gary S. Firestein
    Pages 1-18

  3. T cell receptor rearrangements in arthritis

    • David E. Fox, Nora G. Singer
    Pages 19-53

  4. T cell-independent joint destruction

    • Juliane K. Franz, Thomas Pap, Ulf Müller-Ladner, Renate E. Gay, Gerd R. Burmester, Steffen Gay
    Pages 55-74

  5. Role of T cells in arthritis: Lessons from animal models

    • Wim B. van den Berg
    Pages 75-92

  6. The Th1/Th2 cytokine balance in arthritis

    • Pierre Miossec
    Pages 93-109

  7. Interactions between T cell plasma membranes and monocytes

    • Danielle Burger, Jean-Michel Dayer
    Pages 111-128

  8. Adhesion molecules in arthritis: Control of T cell migration into the synovium

    • Nancy Oppenheimer-Marks, Peter E. Lipsky
    Pages 129-148

  9. T cell reactivity to Epstein-Barr virus in rheumatoid arthritis

    • Marc Bonneville, Emmanuel Scotet, Marie-Alix Peyrat, Annick Lim, Jacques David-Ameline, Elisabeth Houssaint
    Pages 149-167

  10. T cell responses in reactive and Lyme arthritis

    • Joachim Sieper, Jürgen Braun
    Pages 169-187

  11. T cell directed therapies and biologics

    • Ferdinand C. Breedveld
    Pages 189-199

  12. T Cells as primary players in rheumatoid arthritis

    • Michael T. Falta, Brian L. Kotzin
    Pages 201-231

  13. Back Matter

    Pages 233-236
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Keywords

About this book

Rheumatoid arthritis (RA) is the most common and most severe form of inflammatory arthritis. The pathogenesis of RA has been the subject of intense research for several decades. The prevailing hypotheses have changed over the years, and have attempted to incorporate the most recent data. Although T cells represent an important component of the cells which infiltrate the joint synovium, their contribution at a late stage of the disease remains a matter of debate. The goal of this book is to outline the major arguments and data suggesting that T cells may, or may not, be central players in the pathogenesis of chronic RA. While each of the editors and authors has his/her own bias (as will be clear by reading the respective chapters), our hope is that the readers will enjoy a complete and balanced view of the critical questions and experiments. This is not just an intellectual exercise since the direction of future therapeutic interventions depends heavily on how one interprets the pathogenesis of RA and the contribution of T cells.

Editors and Affiliations

  • Departments of Immunology and Rheumatology Hôpital Edouard Herriot, University Claude Bernard, Lyon Cedex 03, France

    Pierre Miossec

  • Division of Rheumatology, UCSD School of Medicine, San Diego, USA

    Gary S. Firestein

  • Department of Rheumatology, University Hospital Nijmegen, Nijmegen, The Netherlands

    Wim B. Berg

Bibliographic Information

  • Book Title: T Cells in Arthritis

  • Editors: Pierre Miossec, Gary S. Firestein, Wim B. Berg

  • Series Title: Progress in Inflammation Research

  • DOI: https://doi.org/10.1007/978-3-0348-8823-3

  • Publisher: Birkhäuser Basel

  • eBook Packages: Springer Book Archive

  • Copyright Information: Springer Basel AG 1998

  • Hardcover ISBN: 978-3-7643-5853-2Published: 21 July 1998

  • Softcover ISBN: 978-3-0348-9787-7Published: 05 November 2012

  • eBook ISBN: 978-3-0348-8823-3Published: 11 March 2013

  • Series ISSN: 1422-7746

  • Series E-ISSN: 2296-4525

  • Edition Number: 1

  • Number of Pages: XII, 236

  • Topics: Medicine/Public Health, general

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