Integration/Interaction of Oncologic Growth

1. Front Matter

   Pages i-xix

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2. Microenvironmental Effects on Tumour Progression and Metastasis

   • Kårstein Måseide, Tuula Kalliomäki, Richard P. Hill

   Pages 1-22

3. Role of Microenvironment on Gene Expression, Angiogenesis and Microvascular Function in Tumors

   • Dai Fukumura

   Pages 23-36

4. Imaging of Angiogenesis In Vivo with Fluorescent Proteins

   • Robert M. Hoffman

   Pages 37-45

5. The Epigenetic Influence of the Tumor Microenvironment on Melanoma Plasticity

   • E.A. Seftor, P.S. Meltzer, D.A. Kirschmann, R.E.B. Seftor, M.J.C. Hendrix

   Pages 47-63

6. Dynamics of Cell Adhesion Interactions during Melanoma Development

   • G. Li, M. Herlyn

   Pages 65-79

7. Matrix Metalloproteinases: Mediators of Tumour-Host Cell Interactions

   • Robert J. Diaz, Alexandra Eichten, Karin E. de Visser, Lisa M. Coussens

   Pages 81-126

8. Role of Brain Microenvironment in Brain Metastases

   • J. Grunfeld, V.K. Puduvalli

   Pages 127-147

9. Influence of the Bone Microenvironment on Breast Cancer Metastasis to Bone

   • B.L. Eckhardt, N. Pouliot, R.L. Anderson

   Pages 149-164

10. Interaction of Tumour with Host Stroma in Hepatocellular Carcinoma

    • D.S.R. Lockwood, D.C. Gotley

    Pages 165-176

11. Dynamic Nature of Tumour-Host Interactions Within the Tumor Microenvironment

    • D. Nagorsen, F. M. Marincola

    Pages 177-187

12. Breast Cancer Progression: A “Vicious Cycle” of Pro-Malignancy Activities is Mediated by Inflammatory Cells, Chemokines and Cytokines

    • A. Ben-Baruch

    Pages 189-217

13. Tumor and Host Endothelial Cell Selective Interactions and Modulation by Microenvironmental Chemokines: Tumor-Endothelial Cell Cross Talk Specificity

    • N. Lamerant-Fayel, C. da Crola Silva, C. Kieda

    Pages 219-231

14. The Role of Chemokine Receptors, in Particular CXCR4, in Lymphoma and Carcinoma Metastasis

    • I.S. Zeelenberg, E. Roos

    Pages 233-244

15. Energy Homeostasis and the Tumor/Host Interaction: The role of the Brain

    • Michael F. Wiater

    Pages 245-258

16. Anti-Angiogenic and Pro-Apoptotic Effects of Dietary Restriction in Experimental Brain Cancer: Role of Glucose and Ketone Bodies

    • T.N. Seyfried, P. Mukherjee

    Pages 259-270

17. Dietary Restriction of Specific Amino Acids Modulates Tumor and Host Interactions

    • G. G. Meadows, Y.-M. Fu

    Pages 271-283

18. Role of Tumour Microenvironment in Chemoresistance

    • Yong Wei, Jessie L.-S. Au

    Pages 285-321

19. Integrin-Mediated Resistance to Chemotherapy-Induced Apoptosis in Cancer Cells

    • R.C. Rintoul, T. Sethi

    Pages 323-333

20. Bone Metastasis Microenvironment Participates in the Development of Androgen Ablation Refractoriness and Chemotherapy Resistance of Prostate Cancer Cells Residing in the Skeleton: Clinical Implications

    • M. Koutsilieris, R. Tenta, D. Tiblalexi, N. Pitulis, E. Sotiriou, P. Lembessis et al.

    Pages 335-344

The present multi-volume Book Series, CANCER GROWTH AND PROGRESSION, encompasses the widest possible framework of cutting edge research in the field of neoplastic pathology and other integrated fields. Normal and pathologic growth is one of the most intensively studied yet challenging areas in pathology. Thus the individual volumes in this series focus on the topics of highest scientific interest for basic and clinical researchers, pathologists, medical and surgical oncologists and allied multidisciplinary teams interested in the study of these aspects of neoplastic growth, progression and inhibition. The range of topics covered is extensive, including but not limited to autonomous growth characteristics of malignancy, phenomena of progression of malignant growth involving the various body systems, and recent advances being made in successful neoplastic inhibition and control. Cell function may be described as producing progression or regression, often found as alternating features in tumors or as variations between normal tissues and tumors. The source of regression in normal melanin producing cells may not be the same as in melanomas. These functions of living matter persist in all phyla of eumetazoans vascular plants as well as in particular species of fungi. However, homo sapiens are the eumetazoan species, which interest us the most. Normal growth processes cannot be entirely understood in all its diversity until we have a thorough knowledge of what constitutes normal growth in various organisms.

• Chemokine
• angiogenesis
• apoptosis
• carcinoma
• chemoresistance
• dynamics
• imaging
• migration

• Washington State University, Pullman, USA

  Gary G. Meadows

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